Atorvastatin - Drug Monograph

Comprehensive information about Atorvastatin including mechanism, indications, dosing, and safety information.

Introduction

Atorvastatin is a synthetic lipid-lowering agent belonging to the HMG-CoA reductase inhibitor class, commonly known as statins. First approved by the FDA in 1996, it has become one of the most widely prescribed medications worldwide for managing dyslipidemia and reducing cardiovascular risk.

Mechanism of Action

Atorvastatin competitively inhibits HMG-CoA reductase, the rate-limiting enzyme in cholesterol biosynthesis in the liver. This inhibition decreases hepatic cholesterol synthesis, resulting in upregulation of LDL receptors on hepatocytes and increased clearance of circulating LDL cholesterol. Atorvastatin also reduces LDL production by inhibiting hepatic VLDL synthesis.

Indications

  • Primary hypercholesterolemia (including heterozygous and homozygous familial hypercholesterolemia)
  • Mixed dyslipidemia
  • Primary prevention of cardiovascular disease in patients with multiple risk factors
  • Secondary prevention of cardiovascular events in patients with established coronary heart disease
  • Pediatric patients (10-17 years) with heterozygous familial hypercholesterolemia

Dosage and Administration

Initial dosing: 10-20 mg orally once daily Maintenance range: 10-80 mg daily Maximum dose: 80 mg daily Special populations:
  • Hepatic impairment: Use with caution; contraindicated in active liver disease
  • Renal impairment: No dosage adjustment necessary
  • Geriatric patients: No dosage adjustment necessary
  • Asian patients: Consider lower starting doses due to increased systemic exposure

Pharmacokinetics

Absorption: Rapidly absorbed with peak plasma concentrations within 1-2 hours; absolute bioavailability approximately 14% Distribution: Highly protein-bound (>98%); volume of distribution ~381 L Metabolism: Extensive hepatic metabolism via CYP3A4 to active ortho- and parahydroxylated metabolites Elimination: Primarily biliary excretion; elimination half-life 14 hours; prolonged inhibitory effect on HMG-CoA reductase due to active metabolites

Contraindications

  • Active liver disease or unexplained persistent elevations of serum transaminases
  • Pregnancy and breastfeeding
  • Hypersensitivity to atorvastatin or any component of the formulation
  • Concomitant use with strong CYP3A4 inhibitors in patients taking high-dose atorvastatin

Warnings and Precautions

Hepatotoxicity: Monitor liver enzymes before initiation and periodically thereafter Myopathy/Rhabdomyolysis: Risk increases with higher doses, advanced age, renal impairment, and concomitant use with certain medications Diabetes: May increase HbA1c and fasting serum glucose levels Cognitive effects: Rare reports of cognitive impairment Avoid in pregnancy: May cause fetal harm

Drug Interactions

Strong CYP3A4 inhibitors: Clarithromycin, itraconazole, HIV protease inhibitors - increase atorvastatin exposure Gemfibrozil: Increases risk of myopathy Cyclosporine: Significantly increases atorvastatin concentrations Oral contraceptives: Increases ethinyl estradiol and norethindrone concentrations Warfarin: May potentiate anticoagulant effect; monitor INR closely Colchicine: Increased risk of myopathy

Adverse Effects

Common (≥2%): Headache, myalgia, arthralgia, diarrhea, nausea, elevated liver enzymes Serious but rare:
  • Rhabdomyolysis with renal failure
  • Severe hepatotoxicity
  • Immune-mediated necrotizing myopathy
  • Hemorrhagic stroke (in patients with recent stroke)
  • Stevens-Johnson syndrome

Monitoring Parameters

  • Lipid panel at baseline, 4-12 weeks after initiation or dose change, and periodically thereafter
  • Liver enzymes at baseline and as clinically indicated
  • CPK levels in patients with muscle symptoms
  • Renal function in patients at risk for rhabdomyolysis
  • HbA1c and fasting glucose in patients with diabetes risk factors
  • Signs and symptoms of myopathy

Patient Education

  • Take atorvastatin at the same time each day, with or without food
  • Report unexplained muscle pain, tenderness, weakness, or brown urine immediately
  • Avoid excessive alcohol consumption
  • Inform all healthcare providers about atorvastatin use
  • Maintain regular follow-up appointments for monitoring
  • Continue lifestyle modifications (diet, exercise)
  • Use effective contraception if of childbearing potential
  • Be aware of potential interactions with other medications

References

1. Grundy SM, et al. 2018 AHA/ACC/AACVPR/AAPA/ABC/ACPM/ADA/AGS/APhA/ASPC/NLA/PCNA Guideline on the Management of Blood Cholesterol. J Am Coll Cardiol. 2019;73(24):e285-e350. 2. Atorvastatin prescribing information. FDA-approved labeling. 3. Thompson PD, et al. Statin-associated side effects. J Am Coll Cardiol. 2016;67(20):2395-2410. 4. Mach F, et al. 2019 ESC/EAS Guidelines for the management of dyslipidaemias. Eur Heart J. 2020;41(1):111-188. 5. Newman CB, et al. Statin Safety and Associated Adverse Events. Arterioscler Thromb Vasc Biol. 2019;39(5):e38-e81.

Medical Disclaimer

The information provided in this article is for educational purposes only and is not intended as a substitute for professional medical advice, diagnosis, or treatment. Always seek the advice of your physician or other qualified health provider with any questions you may have regarding a medical condition. Never disregard professional medical advice or delay in seeking it because of something you have read on this website.

The content on MedQuizzify is designed to support, not replace, the relationship that exists between a patient and their healthcare provider. If you have a medical emergency, please call your doctor or emergency services immediately.

How to Cite This Article

admin. Atorvastatin - Drug Monograph. MedQuizzify [Internet]. 2025 Sep 07 [cited 2025 Sep 07]. Available from: http://medquizzify.pharmacologymentor.com/blog/drug-monograph-atorvastatin

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