Glipizide - Drug Monograph

Comprehensive information about Glipizide including mechanism, indications, dosing, and safety information.

Introduction

Glipizide is a second-generation sulfonylurea oral hypoglycemic agent approved by the FDA in 1984 for the management of type 2 diabetes mellitus. As an insulin secretagogue, it remains a cornerstone therapy in the treatment algorithm for type 2 diabetes when glycemic control cannot be achieved through diet and exercise alone.

Mechanism of Action

Glipizide exerts its primary therapeutic effect by binding to ATP-sensitive potassium channels on pancreatic beta cells. This binding causes depolarization of the cell membrane, opening voltage-dependent calcium channels, and subsequent calcium influx. The increased intracellular calcium concentration stimulates insulin secretion from pancreatic beta cells. Unlike first-generation sulfonylureas, glipizide has minimal effect on cardiovascular potassium channels, potentially reducing cardiovascular side effects.

Indications

  • Primary Indication: Management of type 2 diabetes mellitus as an adjunct to diet and exercise
  • Monotherapy: When glycemic control cannot be achieved by nonpharmacological interventions alone
  • Combination Therapy: May be used with other oral antidiabetic agents (e.g., metformin) or insulin when dual therapy is indicated

Dosage and Administration

Initial Dose: 5 mg once daily, 30 minutes before breakfast Maintenance Dose: May be increased in increments of 2.5-5 mg at weekly intervals Maximum Dose: 40 mg daily (divided doses if >15 mg daily) Special Populations:
  • Geriatric: Start with 2.5 mg daily due to increased hypoglycemia risk
  • Renal Impairment: Use with caution; consider alternative agents in severe impairment
  • Hepatic Impairment: Use with caution; monitor closely for hypoglycemia
Administration: Should be taken 30 minutes before a meal to achieve maximum efficacy

Pharmacokinetics

  • Absorption: Rapid and complete gastrointestinal absorption; bioavailability approaches 100%
  • Distribution: Extensive protein binding (92-99%), primarily to albumin
  • Metabolism: Hepatic metabolism via cytochrome P450 2C9 to inactive metabolites
  • Elimination: Renal excretion (80-90% as metabolites); half-life 2-4 hours
  • Onset/Duration: Onset within 1-3 hours; duration 12-24 hours

Contraindications

  • Type 1 diabetes mellitus
  • Diabetic ketoacidosis
  • Hypersensitivity to glipizide or sulfonylureas
  • Concomitant use with bosentan

Warnings and Precautions

  • Hypoglycemia: Risk increased with renal/hepatic impairment, elderly, malnourished patients
  • Cardiovascular Mortality: Possible increased risk as suggested by UGDP study
  • Hemolytic Anemia: Possible in patients with glucose-6-phosphate dehydrogenase deficiency
  • Syndrome of Inappropriate Antidiuretic Hormone (SIADH): Has been reported
  • Hepatic Porhyria: May precipitate acute attacks in susceptible individuals

Drug Interactions

Significant Interactions:
  • Beta-blockers: Mask hypoglycemic symptoms, potentiate hypoglycemia
  • Alcohol: May cause disulfiram-like reaction and potentiate hypoglycemia
  • Warfarin: Increased anticoagulant effect possible
  • NSAIDs, sulfonamides, MAOIs: May enhance hypoglycemic effect
  • Thiazides, corticosteroids: May decrease hypoglycemic effect
  • CYP2C9 inhibitors/inducers: May affect glipizide concentrations

Adverse Effects

Common (≥1%):
  • Hypoglycemia (dose-related)
  • Gastrointestinal disturbances (nausea, diarrhea, constipation)
  • Dizziness
  • Headache
Serious (<1%):
  • Severe hypoglycemia requiring intervention
  • Hemolytic anemia
  • Hyponatremia/SIADH
  • Hepatic toxicity
  • Disulfiram-like reactions with alcohol
  • Photosensitivity reactions

Monitoring Parameters

  • Glycemic Control: Fasting blood glucose, HbA1c (every 3 months until stable)
  • Hypoglycemia: Regular assessment for signs and symptoms
  • Renal Function: Serum creatinine at baseline and periodically
  • Hepatic Function: Liver enzymes at baseline and as clinically indicated
  • Complete Blood Count: At baseline and if anemia suspected

Patient Education

  • Timing: Take medication 30 minutes before meals
  • Hypoglycemia Recognition: Educate about symptoms (sweating, tremor, confusion)
  • Hypoglycemia Management: Always carry quick-acting glucose source
  • Consistency: Maintain regular meal patterns and exercise schedule
  • Alcohol: Avoid excessive consumption; may cause reaction
  • Sick Days: Contact provider during illness; may need dose adjustment
  • Monitoring: Regular blood glucose monitoring as directed
  • Pregnancy: Use effective contraception; notify provider if planning pregnancy

References

1. American Diabetes Association. (2023). Standards of Medical Care in Diabetes. Diabetes Care. 46(Suppl 1):S1-S291. 2. Glipizide [package insert]. New York, NY: Pfizer; 2022. 3. Inzucchi SE, et al. (2015). Management of Hyperglycemia in Type 2 Diabetes. Diabetes Care. 38(1):140-149. 4. Lexicomp Online. Glipizide: Drug Information. Wolters Kluwer Clinical Drug Information. 5. UK Prospective Diabetes Study (UKPDS) Group. (1998). Lancet. 352(9131):837-853. 6. FDA-approved drug labeling for Glucotrol (glipizide).

Medical Disclaimer

The information provided in this article is for educational purposes only and is not intended as a substitute for professional medical advice, diagnosis, or treatment. Always seek the advice of your physician or other qualified health provider with any questions you may have regarding a medical condition. Never disregard professional medical advice or delay in seeking it because of something you have read on this website.

The content on MedQuizzify is designed to support, not replace, the relationship that exists between a patient and their healthcare provider. If you have a medical emergency, please call your doctor or emergency services immediately.

How to Cite This Article

admin. Glipizide - Drug Monograph. MedQuizzify [Internet]. 2025 Sep 08 [cited 2025 Sep 09]. Available from: http://medquizzify.pharmacologymentor.com/blog/drug-monograph-glipizide

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