N-acetylcysteine - Drug Monograph

Comprehensive information about N-acetylcysteine including mechanism, indications, dosing, and safety information.

Introduction

N-acetylcysteine (NAC) is a modified amino acid that serves as a prodrug to L-cysteine and ultimately contributes to glutathione synthesis. Originally developed as a mucolytic agent, its therapeutic applications have expanded significantly to include acetaminophen overdose management and various off-label uses. NAC represents one of the most versatile pharmacological agents with applications spanning pulmonary, hepatic, psychiatric, and toxicological medicine.

Mechanism of Action

N-acetylcysteine exerts multiple pharmacological effects through distinct mechanisms:

Mucolytic Action: NAC cleaves disulfide bonds in mucoproteins, reducing mucus viscosity and improving clearance from respiratory passages. Antioxidant Activity: As a precursor to L-cysteine, NAC enhances glutathione synthesis, the body's primary endogenous antioxidant. This replenishes glutathione stores depleted during oxidative stress. Acetaminophen Antidote: In overdose, NAC serves as a glutathione substitute, conjugating with the toxic metabolite N-acetyl-p-benzoquinone imine (NAPQI), preventing hepatocellular damage. Anti-inflammatory Effects: NAC modulates inflammatory pathways including NF-κB activation and cytokine production, particularly reducing TNF-α and IL-8. Glutamatergic Modulation: NAC influences glutamate signaling and synaptic plasticity, potentially explaining its benefits in neuropsychiatric conditions.

Indications

FDA-Approved Indications:
  • Acetaminophen overdose (within 8-10 hours of ingestion preferred)
  • Adjuvant mucolytic therapy in acute and chronic bronchopulmonary diseases (chronic bronchitis, emphysema, cystic fibrosis)
Common Off-Label Uses:
  • Prevention of contrast-induced nephropathy
  • Psychiatric disorders (bipolar depression, obsessive-compulsive disorder, addiction)
  • Idiopathic pulmonary fibrosis
  • Chronic obstructive pulmonary disease exacerbations
  • Certain hepatic conditions (non-acetaminophen acute liver failure)

Dosage and Administration

Acetaminophen Overdose:
  • Loading dose: 150 mg/kg in 200 mL D5W over 60 minutes
  • Second dose: 50 mg/kg in 500 mL D5W over 4 hours
  • Third dose: 100 mg/kg in 1000 mL D5W over 16 hours
  • Oral regimen: 140 mg/kg loading, then 70 mg/kg every 4 hours for 17 doses
Mucolytic Therapy:
  • Nebulization: 3-5 mL of 10% solution 3-4 times daily
  • Oral: 600 mg twice daily for chronic conditions
Special Populations:
  • Renal impairment: No dosage adjustment typically needed
  • Hepatic impairment: Use standard dosing with caution in severe impairment
  • Elderly: Consider reduced volume of administration in IV regimens
  • Pediatrics: Similar weight-based dosing for acetaminophen overdose

Pharmacokinetics

Absorption: Oral bioavailability is 6-10% due to significant first-pass metabolism. Peak concentrations occur within 1-2 hours. Distribution: Widely distributed throughout body tissues. Volume of distribution approximately 0.5 L/kg. Crosses placenta and enters breast milk. Metabolism: Extensive hepatic metabolism via deacetylation to cysteine and subsequent incorporation into various metabolic pathways. Elimination: Primarily renal excretion as inactive metabolites. Half-life approximately 5.6 hours. Less than 30% excreted unchanged in urine.

Contraindications

  • Documented hypersensitivity to acetylcysteine or any component of the formulation
  • Patients with asthma and history of bronchospasm with previous NAC administration (relative contraindication)

Warnings and Precautions

Anaphylactoid Reactions: IV administration may cause acute hypersensitivity reactions including flushing, rash, hypotension, and bronchospasm, typically occurring during loading dose. Usually manageable with antihistamines and temporary infusion interruption. Asthma Patients: Use with caution in asthmatic patients due to potential for bronchospasm. Fluid Overload: IV administration contains significant fluid volume—use caution in patients with cardiac failure or renal impairment. Antiemetic Premedication: Consider premedication with antiemetics for IV therapy due to high incidence of nausea/vomiting. Sodium Content: Preparations may contain significant sodium—consider in sodium-restricted patients.

Drug Interactions

Nitroglycerin: Concomitant use may lead to severe hypotension and synergistic vasodilation. Activated Charcoal: May adsorb oral NAC—administer charcoal first if both indicated. Antitussives: Avoid combination as suppression of cough reflex may impair mucus clearance. Certain Antibiotics: NAC may incompletely inactivate some antibiotics (e.g., tetracyclines, aminoglycosides)—separate administration by at least 2 hours.

Adverse Effects

Common (>10%):
  • Nausea, vomiting (especially with IV administration)
  • Rash, flushing
  • Fever
  • Drowsiness
Less Common (1-10%):
  • Bronchospasm (especially in asthmatics)
  • Stomatitis, rhinorrhea
  • Urticaria, pruritus
  • Hypertension or hypotension
Rare (<1%):
  • Anaphylactoid reactions
  • Angioedema
  • Hemorrhage (rare reports of coagulopathy)
  • Arrhythmias

Monitoring Parameters

Acetaminophen Overdose:
  • Serial acetaminophen levels
  • Liver function tests (AST, ALT, bilirubin, INR)
  • Renal function
  • Signs of anaphylactoid reactions during infusion
Chronic Therapy:
  • Pulmonary function tests (if respiratory indication)
  • Symptom assessment
  • Adverse effect monitoring
IV Therapy:
  • Vital signs during infusion
  • Oxygen saturation in respiratory compromised patients
  • Fluid balance in volume-sensitive patients

Patient Education

  • Take oral formulation with food to minimize gastrointestinal upset
  • Report any signs of allergic reaction (rash, itching, swelling)
  • For respiratory indications: maintain adequate hydration to enhance mucolytic effects
  • Do not mix with other medications in nebulizer unless specifically instructed
  • For acetaminophen overdose: understand importance of completing full course despite vomiting
  • Store at room temperature, protected from light
  • Solution may have sulfur-like odor—this is normal and does not indicate spoilage

References

1. Tenenbein M. Benefits of intravenous N-acetylcysteine. Clin Toxicol (Phila). 2015;53(5):417-8. 2. Sadowska AM, et al. Antioxidant and anti-inflammatory efficacy of NAC in the treatment of COPD. Int J Chron Obstruct Pulmon Dis. 2006;1(4):425-34. 3. Prescott LF. Oral or intravenous N-acetylcysteine for acetaminophen poisoning? Ann Emerg Med. 2005;45(4):402-4. 4. Millea PJ. N-acetylcysteine: multiple clinical applications. Am Fam Physician. 2009;80(3):265-9. 5. Drugdex® Micromedex Solutions. N-acetylcysteine. Truven Health Analytics, Inc. Updated 2023. 6. American College of Medical Toxicology. Guidelines for NAC use. J Med Toxicol. 2020;16(2):149-168. 7. Berk M, et al. The promise of N-acetylcysteine in neuropsychiatry. Trends Pharmacol Sci. 2013;34(3):167-77.

Medical Disclaimer

The information provided in this article is for educational purposes only and is not intended as a substitute for professional medical advice, diagnosis, or treatment. Always seek the advice of your physician or other qualified health provider with any questions you may have regarding a medical condition. Never disregard professional medical advice or delay in seeking it because of something you have read on this website.

The content on MedQuizzify is designed to support, not replace, the relationship that exists between a patient and their healthcare provider. If you have a medical emergency, please call your doctor or emergency services immediately.

How to Cite This Article

admin. N-acetylcysteine - Drug Monograph. MedQuizzify [Internet]. 2025 Sep 10 [cited 2025 Sep 10]. Available from: http://medquizzify.pharmacologymentor.com/blog/drug-monograph-n-acetylcysteine

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