Nebivolol - Drug Monograph

Comprehensive information about Nebivolol including mechanism, indications, dosing, and safety information.

Introduction

Nebivolol is a third-generation beta-adrenergic receptor blocker with unique pharmacologic properties that distinguish it from other beta-blockers. It is a racemic mixture of d- and l-nebivolol, with the d-enantiomer primarily responsible for its beta-blocking activity and the l-enantiomer contributing to its vasodilatory effects. Nebivolol is primarily indicated for the management of hypertension and heart failure with reduced ejection fraction.

Mechanism of Action

Nebivolol exhibits highly selective beta-1 adrenergic receptor blockade (approximately 320-fold greater affinity for β1 vs β2 receptors). Its unique mechanism involves endothelial-dependent vasodilation through stimulation of β3-adrenergic receptors, which increases nitric oxide (NO) bioavailability. This dual mechanism results in both reduced cardiac workload (through beta-blockade) and decreased peripheral vascular resistance (through vasodilation).

The drug does not demonstrate intrinsic sympathomimetic activity and has minimal membrane-stabilizing effects. Nebivolol's nitric oxide-mediated vasodilation contributes to its favorable hemodynamic profile, including maintained cardiac output and reduced afterload.

Indications

  • Hypertension: First-line treatment for essential hypertension as monotherapy or in combination with other antihypertensive agents
  • Heart Failure: Treatment of stable chronic heart failure with reduced ejection fraction (NYHA Class II or III) in addition to standard therapy (ACE inhibitors, diuretics)

Dosage and Administration

Hypertension:
  • Initial dose: 5 mg once daily
  • Maintenance dose: 5-40 mg once daily
  • Titration: May increase at 2-week intervals
Heart Failure:
  • Initial dose: 1.25 mg once daily
  • Titration: Double dose every 1-2 weeks as tolerated
  • Target dose: 10 mg once daily
Special Populations:
  • Renal impairment: CrCl <30 mL/min - initial dose 2.5 mg daily
  • Hepatic impairment: Initial dose 2.5 mg daily
  • Geriatric patients: Consider starting with 2.5 mg daily
  • CYP2D6 poor metabolizers: Reduced dose may be necessary

Pharmacokinetics

Absorption: Rapidly absorbed after oral administration with absolute bioavailability of 12% in fast metabolizers and 96% in slow metabolizers Distribution: Extensive tissue distribution with volume of distribution of 673 L; protein binding approximately 98% Metabolism: Extensive hepatic metabolism primarily via CYP2D6 (and to lesser extent CYP2C19 and CYP3A4) with significant first-pass metabolism Elimination: Primarily renal excretion (38% urine, 44% feces) with terminal half-life of 12-19 hours Active metabolites: 4-hydroxynebivolol and glucuronide conjugates contribute to pharmacological activity

Contraindications

  • Severe bradycardia (heart rate <50 bpm)
  • Heart block greater than first degree (without pacemaker)
  • Cardiogenic shock
  • Decompensated cardiac failure
  • Severe hepatic impairment (Child-Pugh Class C)
  • Hypersensitivity to nebivolol or any component of the formulation
  • Sick sinus syndrome (unless pacemaker present)
  • Severe bronchial asthma or severe COPD

Warnings and Precautions

Cardiac Effects: May cause bradycardia, heart block, or worsening heart failure during initiation. Gradually titrate dose and monitor closely. Abrupt Withdrawal: Avoid sudden discontinuation (risk of rebound hypertension, angina exacerbation, or myocardial infarction). Taper over 1-2 weeks. Peripheral Vascular Disease: May precipitate or aggravate symptoms of arterial insufficiency. Diabetes: May mask hypoglycemia symptoms and prolong hypoglycemia. Monitor blood glucose regularly. Thyrotoxicosis: May mask clinical signs of hyperthyroidism. Anesthesia/Surgery: Consider preoperative withdrawal due to impaired ability to respond to reflex stimuli. Bronchospastic Disease: Use caution in patients with chronic bronchitis, emphysema, or asthma.

Drug Interactions

Strong CYP2D6 Inhibitors: Fluoxetine, paroxetine, quinidine - may increase nebivolol concentrations (reduce nebivolol dose) Other Antihypertensives: Additive hypotensive effects with calcium channel blockers, ACE inhibitors, ARBs, diuretics Antiarrhythmics: Increased risk of bradycardia and AV block with digoxin, diltiazem, verapamil Insulin/Oral Hypoglycemics: Enhanced hypoglycemic effects Sympathomimetics: Reduced effectiveness of beta-agonists MAO Inhibitors: Potential for hypertension (avoid concurrent use)

Adverse Effects

Common (>10%):
  • Headache (6-9%)
  • Fatigue (5-8%)
  • Dizziness (2-4%)
  • Bradycardia (2-3%)
Less Common (1-10%):
  • Nausea
  • Diarrhea
  • Dyspnea
  • Insomnia
  • Edema
  • Rash
Serious (<1%):
  • AV block
  • Heart failure exacerbation
  • Bronchospasm
  • Depression
  • Sexual dysfunction
  • Hepatotoxicity

Monitoring Parameters

  • Blood pressure (standing and supine)
  • Heart rate (especially during titration)
  • ECG for signs of conduction abnormalities
  • Renal function (BUN, creatinine)
  • Hepatic function (baseline and periodically)
  • Signs/symptoms of heart failure
  • Blood glucose in diabetic patients
  • Mental status changes
  • Peripheral perfusion

Patient Education

  • Take medication at the same time each day, with or without food
  • Do not stop taking suddenly - consult provider before discontinuing
  • Rise slowly from sitting/lying position to prevent dizziness
  • Monitor pulse rate regularly and report rates below 50 bpm
  • Report unusual fatigue, shortness of breath, weight gain, or swelling
  • Inform all healthcare providers about nebivolol use before procedures
  • Use caution when driving or operating machinery until effects are known
  • Avoid alcohol as it may enhance hypotensive effects
  • Diabetic patients should monitor blood glucose more frequently
  • Notify provider if pregnancy is planned or occurs

References

1. FDA Prescribing Information: Bystolic (nebivolol) tablets 2. Weiss R. Nebivolol: a novel beta-blocker with nitric oxide-induced vasodilation. J Clin Hypertens. 2006;8(1):33-42. 3. Cockcroft JR, et al. Nebivolol vasodilates human forearm vasculature: evidence for an L-arginine/NO-dependent mechanism. J Pharmacol Exp Ther. 1995;274(3):1067-1071. 4. Mangrella M, et al. Nebivolol: a review of its pharmacological and clinical profile. Curr Med Chem. 2008;15(3):242-256. 5. McNeely W, et al. Nebivolol in the management of essential hypertension: a review. Drugs. 1999;57(4):633-651. 6. Cleophas TJ, et al. Nebivolol: a review of its clinical and pharmacological characteristics. Int J Clin Pharmacol Ther. 2006;44(7):344-357. 7. UpToDate: Nebivolol drug information 8. Lexicomp Online: Nebivolol monograph

Medical Disclaimer

The information provided in this article is for educational purposes only and is not intended as a substitute for professional medical advice, diagnosis, or treatment. Always seek the advice of your physician or other qualified health provider with any questions you may have regarding a medical condition. Never disregard professional medical advice or delay in seeking it because of something you have read on this website.

The content on MedQuizzify is designed to support, not replace, the relationship that exists between a patient and their healthcare provider. If you have a medical emergency, please call your doctor or emergency services immediately.

How to Cite This Article

admin. Nebivolol - Drug Monograph. MedQuizzify [Internet]. 2025 Sep 10 [cited 2025 Sep 10]. Available from: http://medquizzify.pharmacologymentor.com/blog/drug-monograph-nebivolol

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